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EGFR — POLDIP2
Text-mined interactions from Literome
Kanda et al., Br J Pharmacol 2001
:
In the present study, we examine the
involvement of
epidermal growth factor (EGF) receptor in thrombin induced
p38 MAPK activation
Funakoshi et al., J Biol Chem 2002
:
Inhibition of
epidermal growth factor-receptor by AG1478
suppressed the AngII induced CREB phosphorylation as well as activation of ERK and
p38MAPK
Seo et al., J Biol Chem 2004
:
UV-induced p38 activation was inhibited by N17 Ras and a farnesyltransferase inhibitor, manumycin A. Gbetagamma also increased the UV-induced phosphorylation of the epidermal growth factor receptor (EGFR), and the UV-induced
p38 activation was blocked by an
EGFR kinase inhibitor, AG1478
Cui et al., Free Radic Biol Med 2006
:
We observed that inhibition of
EGFR kinase activity with AG1478 significantly
attenuated H2O2 induced
p38 ( SAPK ) and ERK1/2 activation, but did not inhibit Akt1 activation
Shin et al., Biochim Biophys Acta 2009
:
Cross interference with TNF-alpha induced TAK1 activation via
EGFR mediated
p38 phosphorylation of TAK1 binding protein 1
Zhou et al., Zhonghua Yi Xue Za Zhi 2012
(MAP Kinase Signaling System) :
The cells were pre treated with antibodies of
ErbB1-4 ,
inhibitors of
p38 mitogen activated protein kinase ( MAPK ), ERK1/2, mitogen- and stress activated protein kinase ( MSK ) 1 and antibody of cAMP-response element binding protein ( CREB )