Hosokawa et al., Clin Exp Immunol 2005
:
On the other hand, we found that not only NF-kappaB, p38 MAPK and ERK but also c-Jun NH2-terminal kinase (JNK) are involved in CCL20 production induced by E. coli LPS
Caruso et al., Gastroenterology 2007
(Inflammatory Bowel Diseases) :
Inhibition of ERK1/2 but not p38 suppressed IL-21 induced MIP-3alpha production
Keates et al., J Immunol 2007
:
MIP-3alpha mediated ERK1/2 activation in Caco-2 cells appeared to require metalloproteinase dependent release of the endogenous EGFR ligand amphiregulin and transactivation of the EGFR
Lee et al., Arch Oral Biol 2008
(Carcinoma, Squamous Cell...) :
Selective inhibitors of p38 and ERK1/2 abolished DFO induced CCL20 expression in both IHOK and HN12 cells, and p38 and ERK1/2 inhibitors prevented DFO induced degradation of I-kappaB and NF-kappaB activation