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HDAC1 — IL1A
Text-mined interactions from Literome
Yoshikawa et al., Arch Otolaryngol Head Neck Surg 2006
(Cholesteatoma, Middle Ear) :
The messenger RNA expressions of
LARC ( liver and activation regulated chemokine ), GMCSF ( granulocyte-macrophage colony stimulating factor ), epiregulin, ICAM1 ( intercellular adhesion molecule 1 ), and TGFA ( transforming growth factor alpha ) were more strongly
up-regulated by
IL-1alpha and/or IL-1beta in MECF than in SF, suggesting that these fibroblasts derived from different tissues retained their typical gene expression profiles
Enya et al., J Leukoc Biol 2008
(Melanoma) :
These results indicate that the reduction in the activity and interaction of
HDAC1 with Sp1 are
critical for the constitutive
IL-1 alpha gene expression
Pietrella et al., Infect Immun 2010
(Inflammation) :
In particular Sap1,
Sap2 , and Sap6 significantly
induced interleukin-1ß (IL-1ß) , tumor necrosis factor alpha ( TNF-a ), and IL-6 production
Wessels et al., Immunobiology 2013
:
The
effects of
histone deacetylase (HDAC)-inhibition by trichostatin A (TSA) on
IL-1ß and TNFa expression and their promoter structures were measured in promyeloid HL-60 cells
Sarlo et al., Cell Immunol 1985
:
The mouse macrophage line P388D1, also could bind SAP and display enhanced
IL-1 production in
response to
SAP
Ku et al., Cytokine 1993
(Carcinoma, Hepatocellular...) :
By contrast, gene expression of the major APR of the mouse,
serum amyloid P-component (SAP) , a structural homologue of CRP, increased in
response to either
IL-1 or IL-6 under the same conditions