◀ Back to IRF1
CXCL10 — IRF1
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Lee et al., J Neuroimmunol 2004
:
In addition, ethanol pretreatment selectively regulated LPS induced NF-kappaB signaling pathway without affecting IFNgamma induced signal transducer and activator of transcription 1 ( STAT1 ) phosphorylation,
interferon regulatory factor-1 (IRF-1) induction or IFNgamma-inducible
IP-10 expression
Buttmann et al., J Vasc Res 2007
:
Interferon-beta is a potent inducer of
interferon regulatory factor-1/2 dependent
IP-10/CXCL10 expression in primary human endothelial cells
Kanda et al., Eur J Immunol 2007
:
Antisense oligonucleotides against NF-kappaB p50, p65, or STAT1 suppressed CXCL9,
CXCL10 , and CXCL11 production, and antisense
IRF-1 suppressed CXCL11 production
Koetzler et al., J Allergy Clin Immunol 2009
(Picornaviridae Infections) :
To define the molecular mechanisms by which NO
inhibits HRV-16 induced epithelial production of
CXCL10 by affecting nuclear translocation and binding of nuclear factor-kappaB (NF-kappaB) and
IFN regulatory factor 1 (IRF-1)
Jauregui et al., Infect Immun 2013
(Bacteroidaceae Infections...) :
Ectopic expression of
IRF-1 in epithelial cells relieved P. gingivalis
induced inhibition of
IP-10 release
Majumder et al., J Immunol 1998
(Fibrosarcoma) :
These results documented the essential role of p48 ( complexed with STAT-1alpha ) for induction and sustained transcription of the IP-10 gene, strongly suggesting that
IRF-1 is not
required for
IP-10 induction by these inflammatory cytokines
Cheng et al., J Interferon Cytokine Res 1998
:
IP-10 gene transcription by virus in astrocytes
requires cooperation of ISRE with adjacent kappaB site but not
IRF-1 or viral transcription ...
IRF-1 is not
required for virus induced
IP-10 gene expression